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Luteinizing Hormone (LH) plays a critical and disrupted role in the pathophysiology of Polycystic Ovary Syndrome (PCOS). In individuals with PCOS, the normal hypothalamic-pituitary-ovarian hormonal feedback loop experiences alterations, leading to an abnormally elevated frequency and amplitude of pulse secretions of luteinizing hormone relative to follicle-stimulating hormone (FSH). This high LH-to-FSH ratio stimulates the ovarian theca cells to produce excessive androgens like testosterone, which subsequently interferes with normal follicular development, inhibits regular ovulation, and contributes directly to the clinical symptoms characteristic of the condition.
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